As shown in Figure 10A, NFATs and their 50 most frequently altered neighboring genes in LUAD were significantly enriched in human T-lymphotropic viruses, type I (HTLV-I) infection; the cGMP-dependent protein kinase, oxytocin, cAMP, T-cell receptor, and Wnt signaling pathways; hepatitis B; long-term potentiation; osteoclast differentiation; and tuberculosis. Here, OXT is linked to tuberculosis.