TGFB1 and myocardial infarction: On the other hand, Shao et al. found that MSC- exosomes could induce cardiomyocyte cell proliferation, suppress their apoptosis, and also target transforming growth factor-beta (TGF-β)-induced transformation of fibroblasts into myofibroblast in myocardial infarction (MI) murine model by up-regulation of miR-29 and miR-24, positively regulating cardiac activities [63].