A study by Zhou et al. (2018) investigated differentially expressed genes between GBM samples and normal brain samples, and found that upregulated genes in a KEGG analysis were enriched in the neuroactive ligand-receptor interaction, cytokine-cytokine receptor interaction and JAK-STAT signaling pathways, implying that some common biological mechanisms exist between the neuroactive ligand receptor interaction and GBM-related pathways. Here, SOAT1 is linked to glioblastoma.