In Figure 2C, they principally participated in B cell receptor signaling pathway, Epstein-Barr virus infection, NF-kappa B signaling pathway, primary immunodeficiency, malaria, hepatocellular carcinoma, tuberculosis, axon guidance, proteasome, PPAR signaling pathway, complement and coagulation cascades, hematopoietic cell lineage, viral protein interaction with cytokine and cytokine receptor, TNF signaling pathway and leukocyte transendothelial migration. This evidence concerns the gene PPARA and inborn error of immunity.