However, following recommendations from the European Leukaemia Network (Döhner et al., 2010), screening for distinct and recurrent molecular drivers of AML, including Nucleophosmin 1 (NPM1), fms like tyrosine kinase 3 (FLT3), and CCAAT/enhancer-binding protein alpha (CEBPA), are currently being used in routine practice, and have significantly improved individual prognosis and clinical management through more targeted therapeutic approaches (Döhner et al., 2017). Here, FLT3 is linked to acute myeloid leukemia.