reported that high Glut1 expression occurred at an early stage of gastric cancer where it facilitates 2-deoxy-2-[18F]fluoro-D-glucose ([18F]FDG) uptake (55), and it is responsible for gastric cancer progression by activating the AKT pathway (56), which plays a key role in glucose metabolism. The gene discussed is SLC2A1; the disease is gastric cancer.