Molecular heterogeneity, proliferation, and metastasis in NSCLC have been associated with various driver mutations in the epidermal growth factor receptor (EGFR), anaplastic lymphoma kinase (ALK), proto-oncogene tyrosine-kinase protein (ROS1), and mesenchymal epithelial transition factor receptor (MET) (1, 2). Here, ALK is linked to non-small cell lung carcinoma.