Similarly, LL-37-exposed cocultures of eosinophils and BEAS-2B cells displayed increased IL-6, IL-8/CXCL-8, and CCL4, while murine LL-37 homolog cRAMP increased bronchial hyperresponsiveness in ovalbumin-induced murine asthma (Jiao et al., 2017). The gene discussed is CXCL8; the disease is asthma.