In murine bleomycin-induced lung fibrosis, inactivation of the FGF10/FGFR2B axis led to increased honeycombing lesions, while FGF10 overexpression enhanced fibrosis resolution (Yuan et al., 2019), further deciphering the complex network of signaling pathways that operate in IPF. This evidence concerns the gene FGF10 and pulmonary fibrosis.