Our results showed that the cat2‐1 mutant was susceptible to B. cinerea B05.10 compared with the wildtype plant, as we previously reported (Yuan et al., 2017), while the expression of CAT2‐N‐SKL driven by the CAT2 promoter did not alter the catalase activity of the cat2‐1 mutant but partially rescued the sensitivity of the mutant to infection (Figure 4a,b). Here, CAT is linked to infection.