Infection with HSV-1 virions lacking pUS9 or with mutations in the KIF5B binding region results in reduced numbers of particles moving anterogradely in cultured sensory neurons and human SK-N-SH neuroblastoma cells, compared to infection with wild-type HSV-1 (Snyder et al., 2008; McGraw et al., 2009; Diefenbach et al., 2016). This evidence concerns the gene KIF5B and infection.