However, our results point in the opposite direction; the increase in CD56+ T cells in BALF coincides with a decreased TH1 alveolitis.5 As the function of CD56+ T cells seems, at least to some degree, to be dependent on the T cell subset and microenvironment,9 we believe that the increased percentage of CD56+ T cells observed in BALF after treatment represents a CD56+ T cell population with a different function than in PB. The gene discussed is NCAM1; the disease is hypersensitivity pneumonitis.