To further clarify the pathogenic genes associated with disease-related FBs, we analysed our scRNA-seq data to explore DEGs that highly expressed in endometriosis FBs, including C3, C7, StAR and S100A10. C3 and C7 were important components in the complement system which participated in inflammation as well as supported tumour growth [20]. The gene discussed is S100A10; the disease is neoplasm.