If this is seen as a sign of inflammation, one could argue that the results obtained in renovascular hypertension will now exclude the possibility that in this form of glomerulonephritis, inflammation will influence afferent renal nerve fibers in spite of our results obtained from neuronal culture exposed to a cytokine such as CXCL1 [9]. The gene discussed is CXCL1; the disease is renovascular hypertension.