Corroborating our observation in humans, murine models of the same genetic alterations show impairments in motion perception (Rendall et al. 2017), together with other sensory deficits (Gabel et al. 2012; Truong et al. 2015; Centanni et al. 2016), suggesting that dyslexics carriers of DCDC2 mutation may constitute a specific subtype of dyslexia where the sensory deficits are a distinctive feature. This evidence concerns the gene DCDC2 and dyslexia.