GYG1 and hypertensive disorder: Further, although we cannot preclude the possibility that mAb HTN-Gn1 induces virus aggregation through cross-linking of virions via available Fabs, the high density of HTN-Gn1 epitopes on the virus surface (Fig. 4 and 5) supports a model whereby both Fab regions of mAb HTN-Gn1 are able to recognize the same virion simultaneously.