The degree of renal fibrosis, assessed through expression of Fibronectin-1 (Figure 3(c)), and the expression of proinflammatory IL-1β (Figure 3(d)) were greater, and the expression of glucose transporter GLUT4 was lower (Figure 3(e)) in DN compared to control mice. The gene discussed is SLC2A4; the disease is liver dysplastic nodule.