IL-8 in other scenarios activates EMT in thyroid cancer cells and hepatocellular carcinoma via overexpression of the AKT/Slug and JAK2/STAT3/Snail1 pathways, respectively [75, 76], and in nasopharyngeal carcinoma through epigenetic silencing of E-cadherin [77]. This evidence concerns the gene CXCL8 and nasopharyngeal carcinoma.