56-treated mice exhibitedincreased levels of brain lysoPS and 20:4 PS lipids, similarly tothe changes observed in ABHD12 knockout mice, although they did notshow any auditory defects, which are typical symptoms of PHARC disease.Moreover, 56-treated and ABHD12 knockout mice exhibiteda heightened immunological responses in a lymphocytic choriomeningitisvirus (LCMV) clone 13 infection animal model, thus highlighting thatABHD12 may have an immunosuppressive function. This evidence concerns the gene ABHD12 and infection.