TAABs were demonstrated to affect (1) early tumor cell clearance (immune surveillance) [36], (2) clonal selection of tumor cells [37,38], (3) cancer promotion through inflammation or post-transcriptional modulation (e.g., anti-p53) [5,39], (4) cancer development through increased oxidative stress in the tumor environment [15]. The gene discussed is TP53; the disease is neoplasm.