Neuroinflammation, reflected by changes in the level of proinflammatory (IL-1β and IL-6) and anti-inflammatory (IL-10) cytokines, as well as the activation of astrocytes and microglia in the hippocampus of affected offspring, may be some of the mechanisms for the behavioral disorders resulting from prenatal exposure to maternal HHC. This evidence concerns the gene IL1B and Atypical behavior.