Although the correlation found between Aβ and tau uptake in the posterior temporoparietal and occipital cortex suggest that DLB patients are associated with AD-related pathology [172], a significant tau burden was present despite minimal amyloid in a small DLB group, suggesting that extensive tauopathy is possible without amyloid deposition in LBD. The gene discussed is MAPT; the disease is Lewy body dementia.