Specifically, it sponges miR-370-3p, which leads to an increase in the levels of its target, NF1. The well-known tumor suppressor NF1 then promotes the hydrolysis of GTP by RAS GTPase, inactivates RAS and negatively regulates the activity of RAS downstream targets, including the RAF/MEK/ERK signaling pathway. Here, NF1 is linked to neoplasm.