FGFR2 and pancreatic intraductal papillary-mucinous neoplasm: In a recent study of 22 pancreatic ITPNs by Basturk et al. based on targeted NGS and/or WES and WGS in a handful of selected cases, alterations in genes related to chromatin remodeling, PI3K signaling, and FGFR2 fusions were found [3], while mutations that are common in IPMN of the pancreas were lacking.