Finally, in COVID-19, the apoptosis of endothelial and epithelial cells in the pulmonary microvasculature and tissues causes the release of chemokines, mainly from the CXC family CXCL9 and CXCL10, MCP-1 (CCL2), CCL2, and CCL3, which can be found in atherosclerotic plaque formation, leading to monocyte/lymphocyte recruitment and infiltration into the subendothelium [23,59,60]. The gene discussed is CXCL10; the disease is COVID-19.