More recently it has been employed as a suppressor of neuroinflammatory response in COVID-19 patients, mainly because of its antiviral properties against SARS-CoV-2 by preventing the entry of the virus into the host cells and inhibition of TNF-α, IL-1β, IL-6, superoxide dismutase, and glutathione peroxidase, and its activation of microglia and astrocytes in neuroinflammatory conditions [283,284,285]. This evidence concerns the gene TNF and COVID-19.