Even though neutrophil recruitment does not depend directly on CCR5 signaling, but on of ELR+ CXC chemokines receptors like CXCR1/2 [38,39], we found that influenza infection increased the percentage of CCR5+ neutrophils present in lung tissue as it was previously reported by Rudd JM, et al. [40]. This evidence concerns the gene CXCR1 and influenza.