KLF5 and systemic sclerosis: Finally, similarly to human SSc BM-MSCs, it has been reported that also BM-MSCs from the KLF5+/−; Fli1+/− murine model of SSc display enhanced migration, proliferation and collagen production in response to TGF-β1, thus showing a preferential differentiation toward myofibroblasts instead of behaving as pericyte precursors [59].