A univariate analysis with two-way ANOVA revealed a total of 81 statistically significant (p value < 0.05) pair-wise relations between the expression levels of 25 BCL2 family members, the mutational status of 26 genes, and the following clinical features: AML FAB subtype, karyotype, tissue type, WBC count, the percentage of blasts in PB and BM, response to treatment (Table S9A). This evidence concerns the gene BCL2 and acute myeloid leukemia.