Three other findings supported this assumption: (1) PrAO substrates, such as benzylamine and methylamine stimulate glucose transport in human adipocytes [37,39]; (2) another phytochemical, resveratrol, is able to inhibit both MAO and lipogenic activities in mouse fat cells [40]; (3) as for caffeine, pharmacological inhibitors of SSAO have been proposed as potential anti-obesity agents [23]. The gene discussed is AOC3; the disease is obesity disorder.