Results from a recent study with an IL-23-induced mouse model of psoriasis have shown that functional neutralization of IL-17C using a specifically generated anti-IL-17C antibody resulted in a) reduced IL-23-induced skin inflammation and b) reduced gene expression of the aforementioned cytokine pattern in the skin, including IL-17A, IL-22, and IL-1β, among others [51]. Here, IL17C is linked to psoriasis.