TNFAIP3 and classic Hodgkin lymphoma: On the other hand, hsa-miR-23a-3p’s inhibitory effect on TNFAIP3, as shown in our study and its manifestation on the cellular level in the tested cHL cell lines, is most probably hampered by various compound heterozygous loss-of-function mutations of TNFAIP3, such as W142STOP codon/del in L-1236, frameshift in amino acid 99/del in KM-H2, or frameshift in amino acid 174/del in HDLM-2 [17].