The presence of proinflammatory cytokines during sepsis allows for the adhesion of activated immune cells to the vascular wall and promotes migration to the surrounding tissues by inducing the expression of molecules, such as selectin E (SEL-E), selectin P (SEL-P), intercellular adhesion molecule 1 (ICAM-1) or vascular adhesion molecule 1 (VCAM-1) [24]. The gene discussed is SELE; the disease is Sepsis.