Altogether, we found that MA caused a concerted pattern of changes in the protein level of HSPAs, an upregulation of the inducible HSPA1 and HSPA6, and downregulation of HSPA2 and HSPA9 encoded by non-stress-inducible genes in MA-sensitive NSCLC cells. The gene discussed is HSPA9; the disease is non-small cell lung carcinoma.