Reasons for impaired gastric motility in patients with LC may be autonomic dysfunction [22], higher postprandial glucose and insulin levels, and a lower secretion of ghrelin [23], a hormone that stimulates gastric emptying and is primarily produced in the stomach, but also to a lesser extent in other organs such as in pancreatic islet cells [36]. Here, INS is linked to laryngotracheoesophageal cleft.