This was proven by the increased cell viability, the transition of the cell cycle from the G1 to the S phase, the upregulation of PCNA and Cyclin D1, and the downregulation of p21. Moreover, BBP modulated the expression of the oncogenic miR-19a/b and PTEN/AKT/p21 axis, revealing that miR-19 plays a crucial role in the promoting effect of BBP on breast cancer cells via the targeting of PTEN 3’UTR. Here, CDKN1A is linked to breast cancer.