Although NE’s main function is to devastate functional proteins of xenobiotics and/or pathogens of various origins, it has also been found to induce deleterious effects on the lungs as elastin-rich connective tissue affording pathologic edematous symptoms such as acute lung injury (ALI), acute respiratory distress syndrome (ARDS), or chronic obstructive pulmonary disorder (COPD) [7,8]. The gene discussed is ELN; the disease is acute respiratory distress syndrome.