GLO1 and posterior cortical atrophy: Although this result needs to be confirmed in a larger sample, it would strongly support, together with our in vitro mechanistic model, the clinical relevance of Glo1/PD-L1/TILs crosstalk in PCa progression through immune surveillance elusion and a role for Glo1 and PD-L1 co-expression to predict patients’ prognosis and response to treatment.