For example, a study published as an abstract showed that peripheral macrophage depletion in mice resulted in reduced 18F-GE180 infarct-related uptake at day 7 compared to control myocardial infarction due to lower CD68+ macrophage content, but increased 68Ga-pentixafor signal because of sustained recruitment of Ly6G+ neutrophils, as well as active calcification of intracavity thrombus detected by 18Na-NaF [68]. Here, CD68 is linked to myocardial infarction.