STAT3 is also responsible for airway remodeling by facilitating epithelial-to-mesenchymal transition [47], involved in both the ASM tissues of patients with asthma and ASM cells under the exposure of asthma-related cytokines and observed to mediate the proliferation, migration, and tube‐forming ability of human lung microvascular endothelial cells [48]. The gene discussed is STAT3; the disease is asthma.