To further identify the potential cell origin of CCL7, we incubated primary VSMCs, adventitial fibroblasts, BMDMs and human umbilical vein endothelial cells (HUVECs) with either Ang II or TNF‐α which could simulate the inflammatory environment in Ang II‐induced AAA,27, 28 Ang II incubation did not alter CCL7 mRNA expression in the cultured cells. The gene discussed is AGT; the disease is triple-A syndrome.