LncRNA NEAT1, methylated with 4 m6A residues, interacted with CYCLINL1 through m6A site and promoted the connection of CYCLINL1 and CDK19.23 In the meantime, NEAT1 selectively bound to the promoter of RUNX2 and recruited CYCLINL1/CDK19 complex on the promoter of RUNX2 in an m6A‐dependent manner, further promoting PCa BM.23 This evidence concerns the gene RUNX2 and posterior cortical atrophy.