Moreover, by inducing a pro-inflammatory and oxidative state [49] and increasing levels of sFas apoptosis [50] and CD14 (bright)/CD16 ( +) monocyte chemoattractant protein [51], the statuses of insulin resistance, dynamic glucose fluctuation, and hyperglycaemia are closely correlated with plaque rupture and result in a poor prognosis in both non-diabetic and diabetic patients with acute MI [52–55]. Here, CD14 is linked to Hyperglycemia.