In addition, to probe the potential mechanisms that Golgi fragmentation as a result of GOLIM4‐L knockdown modulated tumorigenesis in NPC cells, we performed immunoblotting array, revealing that the secretions of multiple chemokines related to tumorigenesis were significantly decreased in the GOLIM4‐L knockdown cells (Figure S10, Supporting Information). The gene discussed is GOLIM4; the disease is nasopharyngeal carcinoma.