AR and Infertility: Because our present study documents the complexity of the disturbances to the androgen-AR signaling axis that result in impaired endometrial receptivity and decidualization processes, future investigation should determine whether elevated androgen levels, increased AR distribution and activity, and/or impaired androgen-AR signaling mechanisms have an impact on uterine-related implantation failure and infertility using uterine tissue/cell-specific AR knockout mice.