NR2F6 and lung adenocarcinoma: miR-142-3p overexpression inhibits the expression of NR2F6, MMP2, and MMP9 and improves caspase-3 expression, thereby inhibiting lung adenocarcinoma cell proliferation, migration, and invasion and enhancing apoptosis, demonstrating that miR-142-3p may be a new therapeutic target for lung adenocarcinoma treatment (Jin et al., 2019).