SLC39A7, also known as ZIP7, is activated by phosphorylation-mediated zinc release from intracellular stores, drives major pathways, such as MAPK, mTOR and PI3K-AKT (30), that are involved in cell survival and proliferation, and it was reported to be overactivated in breast cancer (31) and cervical cancer (32). This evidence concerns the gene SLC39A7 and cervical cancer.