SLC39A7, also known as ZIP7, is activated by phosphorylation-mediated zinc release from intracellular stores, drives major pathways, such as MAPK, mTOR and PI3K-AKT (30), that are involved in cell survival and proliferation, and it was reported to be overactivated in breast cancer (31) and cervical cancer (32). The gene discussed is AKT1; the disease is breast carcinoma.