NFKB1 and neoplasm: NFκB permanent activation maintained the same behavior as previous transcription factors because it diminished at a 100% pro-tumoral phenotype and 20% anti-tumoral phenotypes, creating a perfect balance of tumor elimination and recovery of the tissue due to the secretion of cytotoxic interleukins and interferons (gray areas in Figures 3A and S5, S6 respectively).