Moreover, this notion may not be extensible to all types of AMLs, with the co-occurrence of receptor-type tyrosine-protein phosphatase T (PTPRT), Cullin Associated And Neddylation Dissociated 1(CAND1) and dedicator of cytokinesis 6 (DOCK6) mutations at least in one case of AML with inv.(16) [61]. The gene discussed is CAND1; the disease is acute myeloid leukemia.