Similar patterns of inflammation were detected in patients with severe COVID-19, where there is a state of hyper-inflammation [23], with an increase in the levels of interferon-γ, TNF-α, C-Reactive Protein (CRP) and cytokines, in particular, IL-10, IL-6, and IL-17, which correlates with a significant reduction in T cells population, and even the surviving T cells are functionally exhausted with impaired proliferation [24–26]. The gene discussed is CRP; the disease is COVID-19.