Morpholino-mediated knock down of Cfap161 in zebrafish led to the loss of outer dynein arms, reduced beating frequency of pronephric cilia and strong ciliopathy phenotypes including left–right asymmetry defects in one study37, but only a curved body axis and hydrocephalus in a subsequent analysis38 leaving some uncertainties concerning the ciliary function of CFAP161. This evidence concerns the gene CFAP161 and ciliopathy.